Exercise, Food, Medical, New


see wikipedia (http://en.wikipedia.org/wiki/Ribose) where they state

“Ribose is an organic compound with the formula C5H10O5; specifically, a monosaccharide (simple sugar) with linear form H−(C=O)−(CHOH)4−H, which has all the hydroxyl groups on the same side in the Fischer projection.”

=== me ===

Ribose is used by those suffering from Chronic Fatigue Syndrome to boost their energy. Ribose is a precursor to ATP.

How is ribose metabolized? See (http://nashua.case.edu/PathwaysBiocyc/Web/?viewID=5f1d491c-b24a-4988-af1a-201164ea2240&pwgid=01a8c4ee-8f08-45cc-ba1c-258ab2a6964c&terms=&type=Contains&page=1) for details.


Exercise, Medical, New, Vitamin


from wikipedia (http://en.wikipedia.org/wiki/Beta-alanine) where they state

β-Alanine is the rate-limiting precursor of carnosine, which is to say carnosine levels are limited by the amount of available β-alanine. Supplementation with β-alanine has been shown to increase the concentration of carnosine in muscles, decrease fatigue in athletes and increase total muscular work done.

see also (http://fitness.mercola.com/sites/fitness/archive/2011/09/11/why-muscles-are-sore-after-workouts.aspx) where they discuss this.



Exercise, Food, Medical, New

Rhodiola Rosea

Phytomedicine. 2011 Feb 15;18(4):235-44. doi: 10.1016/j.phymed.2010.08.014. Epub 2010 Oct 30.

The effectiveness and efficacy of Rhodiola rosea L.: a systematic review of randomized clinical trials.


Complementary Medicine, PCMD, University of Exeter, UK. shaohung@pms.ac.uk



To critically assess the current evidence from randomized clinical trials (RCTs) for or against the effectiveness or efficacy of Rhodiola rosea.


Systematic literature searches were performed in six electronic databases: AMED (1985-July 2009), CINAHL (1982-July 2009), The Cochrane Library (search in July 2009), EMBASE (1974-July 2009), MEDLINE (1950-July 2009) and Web of Science (searched in July 2009). No language restrictions were imposed. Reference lists of all retrieved articles were searched, and experts and manufacturers were contacted for unpublished RCT.


RCTs testing the efficacy or effectiveness of mono-preparations of R. rosea as sole treatment administered orally against a control intervention in any human individual suffering from any condition or healthy human volunteers were included. Studies were selected, data extracted, and quality assessed by two independent reviewers.


Eleven RCTs met the inclusion criteria; all were placebo-controlled. Six trials investigated the effects of R. rosea on physical performance, four on mental performance, and two in patients diagnosed with mental health condition. The methodological quality of most trials was moderate or good. Only few mild adverse events were reported.


R. rosea may have beneficial effects on physical performance, mental performance, and certain mental health conditions. There is, however, a lack of independent replications of the single different studies. Five of the 10 RCTs reached more than three points on the Jadad score (i.e., good quality). More research seems warranted.

Copyright © 2010 Elsevier GmbH. All rights reserved.

[PubMed – indexed for MEDLINE]
New, Web Sites

Web Site: National Geographic


These folks do run some nutrition articles. Here are a few with links:


Cocaine Addiction Uses Same Brain Paths as Salt Cravings


Why Are We So Fat?


Can Sugar Make You Stupid? “High Concern” in Wake of Rat Study


Medical, New

Meat consumption and mortality – results from the European Prospective Investigation into Cancer and Nutrition

Meat consumption and mortality – results from the European Prospective Investigation into Cancer and Nutrition

from (http://www.biomedcentral.com/1741-7015/11/63/abstract)

Where they report in part:



Recently, some US cohorts have shown a moderate association between red and processed meat consumption and mortality supporting the results of previous studies among vegetarians. The aim of this study was to examine the association of red meat, processed meat, and poultry consumption with the risk of early death in the European Prospective Investigation into Cancer and Nutrition (EPIC).


Included in the analysis were 448,568 men and women without prevalent cancer, stroke, or myocardial infarction, and with complete information on diet, smoking, physical activity and body mass index, who were between 35 and 69 years old at baseline. Cox proportional hazards regression was used to examine the association of meat consumption with all-cause and cause-specific mortality.


As of June 2009, 26,344 deaths were observed. After multivariate adjustment, a high consumption of red meat was related to higher all-cause mortality (hazard ratio (HR) = 1.14, 95% confidence interval (CI) 1.01 to 1.28, 160+ versus 10 to 19.9 g/day), and the association was stronger for processed meat (HR = 1.44, 95% CI 1.24 to 1.66, 160+ versus 10 to 19.9 g/day). After correction for measurement error, higher all-cause mortality remained significant only for processed meat (HR = 1.18, 95% CI 1.11 to 1.25, per 50 g/d). We estimated that 3.3% (95% CI 1.5% to 5.0%) of deaths could be prevented if all participants had a processed meat consumption of less than 20 g/day. Significant associations with processed meat intake were observed for cardiovascular diseases, cancer, and ‘other causes of death’. The consumption of poultry was not related to all-cause mortality.


The results of our analysis support a moderate positive association between processed meat consumption and mortality, in particular due to cardiovascular diseases, but also to cancer.


diet; meat; mortality; cohort; Europe; cardiovascular; cancer

Medical, New

Intense Sweetness Surpasses Cocaine Reward

Intense Sweetness Surpasses Cocaine Reward

from (http://www.plosone.org/article/fetchArticle.action?articleURI=info%3Adoi%2F10.1371%2Fjournal.pone.0000698)

where they state in part:


“Our findings clearly demonstrate that intense sweetness can surpass cocaine reward, even in drug-sensitized and -addicted individuals. We speculate that the addictive potential of intense sweetness results from an inborn hypersensitivity to sweet tastants. In most mammals, including rats and humans, sweet receptors evolved in ancestral environments poor in sugars and are thus not adapted to high concentrations of sweet tastants. The supranormal stimulation of these receptors by sugar-rich diets, such as those now widely available in modern societies, would generate a supranormal reward signal in the brain, with the potential to override self-control mechanisms and thus to lead to addiction.”

[-] Sources and References